That is very interesting. If genes in the mitochondria are defect from aging, just inserting it into the nucleus seems a very nice idea. However, mitochondrial defects will happen randomly, so every cell would need another protein repaired? Or is the basic idea expressing all vital mt-genes in the nucleus, so defects don't be lethal to the cells anymore?
Gotta dig into it, it seems :D
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[DIYbio] Re: Longecity: a crowdfunding strategy worth watching
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